---
module: 049-02
language: en
chapter: 49
title: "Gastrointestinal History, Examination, Investigations, and Acute Presentations"
module_title: "Bedside abdominal reasoning, physiological consequences, and time-critical investigation"
source_sha256: 0da3ce892f5b437308d7938c46ef728a9c813b01997630a0018d54ca5cacd3c2
---
# Bedside abdominal reasoning and time-critical tests

## History as a physiological map
### Pain onset and movement trace inflammation spread
### Vomit content estimates level and duration
### Stool features separate bleeding, inflammation, malabsorption
### Urgency: perfusion, sepsis, peritonism, organ failure

## Pain mechanisms and history
### Visceral pain poorly localised
#### Afferents converge across spinal segments
#### Foregut epigastric, midgut periumbilical, hindgut lower
### Parietal peritoneum: sharp, local, worse on cough
#### Central to focal migration reflects this shift
#### Muted in older, immunosuppressed, pregnant patients
### Sudden maximal pain: perforation, occlusion, rupture
### Colic: contraction against resistance
#### Biliary pain often steady after onset
### Pain out of proportion suggests mesenteric ischaemia
#### Mucosa hypoxic before peritoneum inflamed
### Give analgesia promptly, examine repeatedly
### Record timing, radiation, pretest modifiers
#### Improvement alone does not exclude danger

## Consequences of vomiting
### Gastric acid loss gives metabolic alkalosis
#### Volume contraction drives sodium retention
#### Distal potassium and hydrogen loss follows
### Low potassium impairs motility and conduction
#### Worsens obstruction and vomiting
### Distal bowel loss of bicarbonate gives acidosis
### Replace chloride and potassium, not only water
### Stopped vomiting is not resolved obstruction

## Bleeding severity and resuscitation
### Colour does not indicate severity
#### Coffee grounds can coexist with active bleeding
#### Melaena may come from small bowel or right colon
#### Brisk upper bleeding can give bright rectal blood
### First haemoglobin may be normal
### Perfusion signs and ongoing loss guide resuscitation
### Haemoglobin fall also reflects dilution
### Source, haemostasis, restarting antithrombotics separate

## Examination
### Starts before touch
#### Motionless patient suggests peritoneal irritation
#### Restlessness more typical of colic
### Resuscitate first if shock or altered consciousness
### Voluntary guarding eases, rigidity persists
### Rebound testing unnecessary and painful
### Pulsatile mass with shock: do not press
### Bowel sounds have low discriminatory accuracy
#### Distension, obstipation, imaging more useful

## Laboratory tests and pregnancy
### Tests measure consequences and competing mechanisms
### Lipase supports pancreatitis only in a compatible syndrome
### Normal lactate possible in early ischaemia
#### Biomarker never overrides high-risk anatomy
### Pregnancy testing whenever biologically possible
#### Do not rely on relationship status alone
#### Uterus displaces organs, signs differ
#### Needed imaging not delayed by fear

## Imaging and endoscopy
### Imaging follows the question and stability
### Ultrasound fast but operator and gas dependent
### Computed tomography finds transition points
### Angiography urgent: portal phase misses vessels
### Uncontrolled bleeding may need theatre instead
### Endoscopy needs physiological preparation
#### Bowel preparation worsens dehydration, electrolytes
#### Full colonoscopy in severe colitis risks perforation
#### Capsule may lodge, patency assessment reduces risk

## Obstruction and perforation
### Mechanical or functional, simple or strangulated
### Closed loop obstructed at two points
#### Distension compromises venous then arterial flow
### Distension creates a self-worsening cycle
#### Barrier fails, translocation, necrosis, perforation
### Colicky to constant pain demands reassessment
### Perforation releases gas and contents
#### Free gas absent in contained or small leaks
#### Question is controllable contamination and viability

## Mimics and serial reassessment
### Extra-abdominal causes: myocardial, pneumonia, ketoacidosis
### Fluid response does not prove the problem solved
### Compare the same sites with time and examiner
### A soft abdomen does not exclude ischaemia

## Safe discharge
### Needs more than pain improvement
### Explicit return triggers explained
### Owners assigned for pending results
### Teach-back of warning signs and plan
### Discharge transfers surveillance to the community
